[1] Thorburn G D. The placenta, prostaglandins and parturition: a review[J]. Reprod Fert Dev, 1991, 3(3): 277-294.[2] Sugimoto Y, Narumiya S, Ichikawa A. Distribution and function of prostanoid receptors: studies from knockout mice[J]. Prog Lipid Res, 2000, 39(4): 289-314.[3] Narumiya S, Sugimoto Y, Ushikubi F. Prostanoid receptors: structures, properties, and functions[J]. Phys Rev, 1999, 79(4): 1 193-1 226.[4] Kreydiyyeh S I, Riman S, Serhan M, et al. TNF-alpha modulates hepatic Na+K+ ATPase activity via PGE2 and EP2 receptors[J]. Prostag Oth Lipid Med, 2007, 83(4): 295-303.[5] Kimura M, Osumi S, Ogihara M. Prostaglandin E-2 (EP1) receptor agonist-induced DNA synthesis and proliferation in primary cultures of adult rat hepatocytes: The involvement of TGF-alpha[J]. Endocrinology, 2001, 142(10): 4 428-4 440.[6] Mater M K, Thelen A P, Jump D B. Arachidonic acid and PGE(2) regulation of hepatic lipogenic gene expression[J]. J Lipid Research, 1999, 40(6): 1 045-1 052.[7] Kuhnz W, Hoyer G A, Backhus S. Identification of the major metabolites of the prostaglandin E2-analogue sulprostone in human plasma, and isolation from urine (in vivo) and liver perfusate (in vitro) of female guinea-pigs[J]. Drug Metab Dispos, 1991, 19(5): 920-925.[8] Liu Cheng-quan(刘承权), Chen Gen-fu(陈根富), Hu Yan(胡炎), et al. A pharmacodynamic study on sulprostone(磺前列酮的药效学研究)[J]. Chin Pharmacol Bull(中国药理学通报), 1992, 6(1): 471-474.[9] Zhao Xiu-ju(赵秀举),Wang Yu-lan(王玉兰). Applications of NMR-based metabonomic approaches in the assessment of drug toxicity(代谢组NMR分析与药物毒理研究)[J]. Chinese J Magn Reson(波谱学杂志) 2011, 28(1): 1-17.[10] Waters N J, Garrod S, Farrant R D, et al. High-resolution magic angle spinning 1]H NMR spectroscopy of intact liver and kidney: Optimization of sample preparation procedures and biochemical stability of tissue during spectral acquisition[J]. Anal Biochem, 2000, 282(1): 16-23.[11] vanBaal J, deJong M D, Zijlstra F J, et al. Endogenously produced prostanoids stimulate calcium reabsorption in the rabbit cortical collecting system[J]. J Physiol-London, 1996, 497(1): 229-239.[12] Liu M L, Nicholson J K, London J C. High-resolution diffusion and relaxation edited one- and two-dimensional 1H NMR- spectroscopy of biological fluids[J]. Anal Chem, 1996, 68(19): 3 370-3 376.[13] Tang H R, Wang Y L, Nicholson J K. Use of relaxation-edited one-dimensional and two dimensional nuclear magnetic resonance spectroscopy to improve detection of small metabolites in blood plasma[J]. Anal Biochem, 2004, 325(2): 260-272.[14] Xiao Chao-ni(肖超妮),Liu Hong-bing(刘红兵),Dai Hui(戴惠),et al. Efficient Identification and structural elucidation of metabolites using HPLC-DAD-SPE-CryoNMRMS method(基于HPLC-DAD-SPE-CryoNMR-MS技术的代谢物快速定性和结构确定)[J]. Chinese J Magn Reson(波谱学杂志), 2009, 26(1): 1-16.[15] Zhang L M, Ye Y F, An Y P, et al. Systems responses of rats to aflatoxin B1 exposure revealed with metabonomic changes in multiple biological matrices[J]. J Proteome Res, 2011, 10(2): 614-623.[16] Fan W M T. Metabolite profiling by one- and two-dimensional NMR analysis of complex mixtures[J]. Prog Nucl Magn Reson Spect, 1996, 28: 161-219.[17] Nicholson J K, Foxall P J D, Spraul M, et al. 750 MHz 1H and 1H-13C NMR spectroscopy of human blood plasma[J]. Anal Chem, 1995, 67(5): 793-811.[18] Puschel G P, Kirchner C, Schroder A, et al. Glycogenolytic and antiglycogenolytic prostaglandin E2 actions in rat hepatocytes are mediated via different signalling pathways[J]. Eur J Biochem, 1993, 218(3): 1 083-1 089.[19] Mine T, Kojima I, Ogata E. Mechanism of prostaglandin E2-induced glucose production in rat hepatocytes[J]. Endocrinology, 1990, 126(6): 2 831-2 836.[20] Puschel G P, Christ B. Inhibition by PGE2 of glucagon-induced increase in phosphoenolpyruvate carboxykinase mRNA and acceleration of mRNA degradation in cultured rat hepatocytes[J]. Febs Lett, 1994, 351(3): 353-356.[21] Haussinger D, Stehle T, Tranthi T A, et al. Prostaglandin responses in isolated perfused rat liver: Ca2+ and K+ fluxes, hemodynamic and metabolic effects[J]. Biol Chem HoppeSeyler, 1987, 368(11): 1 509-1 513.[22] Iwai M, Gardemann A, Puschel G, et al. Potential role for prostaglandin F2α, D2, E2 and thromboxane A2 in mediating the metabolic and hemodynamic actions of sympathetic nerves in perfused rat liver[J]. Eur J Biochem, 1988, 175(1): 45-50.[23] Okumura T, Saito K. Effect of prostaglandins on glycogenesis and glycogenolysis in primary cultures of rathepatocytes: Arole of prostaglandin D2 in the liver[J]. Prostaglandins, 1990, 39(5): 525-540.[24] Okumura T, Saito K. A sex difference in the effect of prostaglandins on hormone-stimulated glycogenolysis in primary cultures of rat hepatocytes[J]. Biochim Biophys Acta, 1990, 1 051(3): 300-305.[25] Ames B N, Elson-Schwab I, Silver E A. Highdose vitamin therapy stimulates variant enzymes with decreased coenzyme binding affinity (increased K-m): relevance to genetic disease and polymorphisms[J]. Am J Clin Nutr, 2002, 75(4): 616-658.[26] Kojima M, Morisaki T, Izuhara K, et al. Lipopolysaccharide increases cyclo-oxygenase-2 expression in a colon carcinoma cell line through nuclear factor-kappa B activation[J]. Oncogene, 2000, 19(9): 1 225-1 231.[27] Johnson L R, Guthrie P. Effect of cholecystokinin and 16,16-dimethyl prostaglandin E2 on RNA and DNA of gastric and duodenal mucosa[J]. Gastroenterology, 1976, 70(1): 59-65.[28] Smith K L, Tisdale M J. Mechanism of muscle protein degradation in cancer cachexia[J]. British J Cancer, 1993, 68(2): 314-318.[29] Tisdale M J. Inhibition of lipolysis and muscle protein degradation by EPA in cancer cachexia[J]. Nutrition, 1996, 12(1): S31-S33.[30] Kitada H, Miyata M, Nakamura T, et al. Protective role of hydroxysteroid sulfotransferase in lithocholic acid-induced liver toxicity[J]. J Biol Chem, 2003, 278(20): 17 838-17 844.[31] Ljubuncic P, Tanne Z, Bomzon A. Evidence of a systemic phenomenon for oxidative stress in cholestatic liver disease[J]. Gut, 2000, 47(5): 710-716.[32] Jaeschke H, Farhood A. Neutrophil and Kupffer cell-induced oxidant stress and ischemia-reperfusion injury in rat liver[J]. Am J Physiol, 1991, 260(3): G355-G362.[33] Masubuchi Y, Suda C, Horie T. Involvement of mitochondrial permeability transition in acetaminophen-induced liver injury in mice[J]. J Hepatol, 2005, 42(1): 110-116. |